Articles
Minamata-Table-02
Table 4
Chronographical onset of Minamata disease
Year Congenital Infant Adult Total 1953 1 1 1954 5 7 12 1955 5 2 8 15 1956 7 21 23 51 1957 6 2 8 1958 2 3 5 1959 3 1 16 20 1960 4 4 Total 23 30 63 116 Mercury contents (ppm) in the hair of the patient of congenital Minamata disease and of their mothers
No. Birth date Patient Mother
1959-61 1963 1966 1960-61 1961-63 1 1956 7 59.0 12.0 3.56 72.9 6.0 2 1957 2 34.3 6.0 9.0 65.4 8.0 3 1957 5 61.9 39.0 14.0 101.0 4 1957 5 4.0 5.0 5 1956 4 51.8 18.0 5.0 6 1956 5 57.8 191.0 32.0 7 1957 10 37.7 54.0 3.5 8 1956 7 26.2 7.0 9.35 4.4 9 1958 9 35.0 10 1958 11 5.25 1.0 6.5 2.0 11 1955 8 11.3 12.0 12.7 12 1956 4 8.14 7.0 2.5 1.8 13 1956 6 10.6 14 1955 11 100.0 90.0 15 1955 8 73.5 19.0 5.79 16.0 16 1956 12 14.9 13.0 10.1 7.03 10.0 17 1955 1 6.0 4.0 5.1 19 1957 8 29.0 32.0* 22 1959 9 28.0 0.0
Note:
1. Cases 6 and 7 were sisters.
2. * measured in 1963.Comparison in pathology of Minamata disease among the congenital, non-fetal, and adult cases (Takeuchi, 1968)
Pathological features Congenital Infantile Adult 1 Cortical disturbance of cerebrum +++ +++ +++ 2 Cerebellar disturbance of granular cell type +++ +++ +++ 3 Central type of granular atropy + ++ ++ 4 Intense reduction of brain +++ +++~++ + 5 Wide distribution of lesions +++ +++ + 6 Preferential localization of lesions ± ++ +++ 7 Degree of the granular cell disturbance + +++ +++ 8 Hypoplastic change of cytoarchitecture* +++ - - 9 Malformation of neurons +++ - -
*: remaining matrix cells, evidence of nerve cells in cerebral medulla, abnormal cytoarchitecture (columnar block, etc.), hypoplastic narrowing of granular layer in cerebellum, and hypoplastic corpus callosum
On Reality. Publisher and editor: Bo Walhjalt. ISSN 1650-9323.
© Bo Walhjalt and authors. | Comments on this page
Latest update 2002-12-05original url of this page: http://www.gbg.bonet.se/bwf/art/har/ts2.html