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REFERENCES

  1. Harada, Y. (1968a)
    Infantile Minamata disease. Minamata Disease, (Study Group of Minamata Disease, Kumamoto University ed.), Kumamoto University, Kumamoto, 73-91.
  2. Harada, Y. (1968b)
    Congenital Minamata disease. Minamata Disease, (Study Group of Minamata Disease, Kumamoto University ed.), Kumamoto University, Kumamoto, 93-117.
  3. Harada, Y. and Moriyama, H. (1976)
    Congenital Minamada disease. Bull. Inst. Const. Med. Kumamoto Univ., 26, Supplement: 1-57.
  4. Hunter, D. and Russell, D.S. (1954)
    Focal cerebral and cerebellar atrophy in a human subject due to organic mercury compounds. J. Neurol. Neurosurg. Psychiatry, 17: 235-241.
  5. Irukayama, K., Kai, F., Fujiki, M. and Kondo, T. (1962)
    Studies on the origin of the causative agent of Minamata disease. III. Industrial wastes containing mercury compounds from Minamata Factory. Kumamoto Med. J., 15: 57-68.
  6. Kondo, T. (1964)
    Studies on the course that the causative agent of Minamata disease was formed, especially on the organomercury compound extracted from the acetaldehyde plant in Minamata Factory and organomercury compound contained in the shellfish from Minamata Bay. J. Kumamoto Med. Soc., 38: 353-373 (in Japanese).
  7. Nagano, S., Kida, T., Harada, Y., Hashiguchi, K., Ueno, T., Kumabe, H., Nagayoshi, T., Setomoto, S., Inoguchi, K., Ueda, K., Ikesawa, A., Nishida, S., Kawahara, K., Ninomiya, T., Miyatake, K., Sakaguchi, T., Kakita, T., and Fuke, M. (1960)
    Study on the infantile Minamata disease. J. Kumamoto Med. Soc., 34, Suppl III: 511-520 (Japanese).
  8. Takeuchi, T. (1968)
    Minamata disease in adult. Minamata disease, (Study Group of Minamata Disease, Kumamoto University ed.), Kumamoto University, Kumamoto, 141-228.
  9. Tokuomi, A. (1968)
    Minamata disease in adult. Minamata Disease, (Study Group of Minamata Disease, Kumamoto University ed.), Kumamoto University, Kumamoto, 37-72.
  10. Uchida, M., Hirakawa, K. and Inoue, T. (1961)
    Biochemical studies on Minamata disease. III. Relationships between the causal agent of the disease and the mercury compound in the shellfish with reference to their chemical behaviors. Kumamoto Med. J., 14: 171-179.

 

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